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Oligomerization and dissociation of AP-1 adaptors are regulated by cargo signals and by ArfGAP1-induced GTP hydrolysis.

Title: Oligomerization and dissociation of AP-1 adaptors are regulated by cargo signals and by ArfGAP1-induced GTP hydrolysis.
Authors: Meyer DM; Biozentrum, University of Basel, CH-4056 Basel, Switzerland.; Crottet P; Maco B; Degtyar E; Cassel D; Spiess M
Source: Molecular biology of the cell [Mol Biol Cell] 2005 Oct; Vol. 16 (10), pp. 4745-54. Date of Electronic Publication: 2005 Aug 10.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Language: English
Journal Info: Publisher: American Society for Cell Biology Country of Publication: United States NLM ID: 9201390 Publication Model: Print-Electronic Cited Medium: Print ISSN: 1059-1524 (Print) Linking ISSN: 10591524 NLM ISO Abbreviation: Mol Biol Cell Subsets: MEDLINE
Imprint Name(s): Original Publication: Bethesda, MD : American Society for Cell Biology, c1992-
MeSH Terms: GTPase-Activating Proteins/*metabolism ; Guanosine Triphosphate/*metabolism ; Protein Sorting Signals/*physiology ; Transcription Factor AP-1/*metabolism; Adaptor Protein Complex 2/metabolism ; Cell Membrane/metabolism ; Clathrin/metabolism ; Clathrin-Coated Vesicles/metabolism ; Cytosol/metabolism ; Liposomes/metabolism ; Animals ; COS Cells ; Cattle ; Chlorocebus aethiops ; Hydrolysis ; In Vitro Techniques ; Molecular Weight
Abstract: The mechanism of AP-1/clathrin coat formation was analyzed using purified adaptor proteins and synthetic liposomes presenting tyrosine sorting signals. AP-1 adaptors recruited in the presence of Arf1.GTP and sorting signals were found to oligomerize to high-molecular-weight complexes even in the absence of clathrin. The appendage domains of the AP-1 adaptins were not required for oligomerization. On GTP hydrolysis induced by the GTPase-activating protein ArfGAP1, the complexes were disassembled and AP-1 dissociated from the membrane. AP-1 stimulated ArfGAP1 activity, suggesting a role of AP-1 in the regulation of the Arf1 "GTPase timer." In the presence of cytosol, AP-1 could be recruited to liposomes without sorting signals, consistent with the existence of docking factors in the cytosol. Under these conditions, however, AP-1 remained monomeric, and recruitment in the presence of GTP was short-lived. Sorting signals allowed stable recruitment and oligomerization also in the presence of cytosol. These results suggest a mechanism whereby initial assembly of AP-1 with Arf1.GTP and ArfGAP1 on the membrane stimulates Arf1 GTPase activity, whereas interaction with cargo induces oligomerization and reduces the rate of GTP hydrolysis, thus contributing to efficient cargo sorting.
References: Proc Natl Acad Sci U S A. 1999 Feb 16;96(4):1224-8. (PMID: 9990005); EMBO J. 1999 Feb 1;18(3):555-64. (PMID: 9927415); Cell. 1999 Mar 19;96(6):893-902. (PMID: 10102276); Proc Natl Acad Sci U S A. 1999 Apr 27;96(9):5013-8. (PMID: 10220410); Science. 1999 Aug 20;285(5431):1268-71. (PMID: 10455054); EMBO J. 1999 Sep 15;18(18):4935-48. (PMID: 10487746); J Cell Biol. 2005 Jan 17;168(2):281-90. (PMID: 15657398); J Biol Chem. 2005 Jun 3;280(22):21661-6. (PMID: 15802264); J Cell Sci. 2000 Jan;113 ( Pt 1):135-44. (PMID: 10591632); FEBS Lett. 1999 Dec 3;462(3):267-72. (PMID: 10622709); Cell. 2000 Mar 17;100(6):671-9. (PMID: 10761932); Eur J Cell Biol. 2000 May;79(5):336-42. (PMID: 10887964); J Biol Chem. 2000 Aug 4;275(31):23615-9. (PMID: 10811810); Mol Biol Cell. 2000 Nov;11(11):3723-36. (PMID: 11071902); Cell. 2000 Nov 10;103(4):569-81. (PMID: 11106728); J Cell Sci. 2001 Jan;114(Pt 2):353-65. (PMID: 11148137); Methods Enzymol. 2001;329:307-16. (PMID: 11210549); Nat Rev Mol Cell Biol. 2000 Dec;1(3):187-98. (PMID: 11252894); Nat Cell Biol. 2001 Jun;3(6):531-7. (PMID: 11389436); Curr Opin Cell Biol. 2001 Aug;13(4):444-53. (PMID: 11454451); J Biol Chem. 2001 Dec 21;276(51):47834-9. (PMID: 11592960); J Cell Biol. 2001 Dec 24;155(7):1199-212. (PMID: 11748249); J Biol Chem. 2002 Mar 15;277(11):9468-73. (PMID: 11777925); Traffic. 2002 Aug;3(8):537-46. (PMID: 12121417); J Cell Biol. 2002 Oct 14;159(1):69-78. (PMID: 12379802); Mol Biol Cell. 2002 Oct;13(10):3672-82. (PMID: 12388765); Eur J Cell Biol. 2002 Dec;81(12):647-53. (PMID: 12553665); Traffic. 2003 Feb;4(2):65-73. (PMID: 12559033); Mol Biol Cell. 2003 Feb;14(2):625-41. (PMID: 12589059); J Biol Chem. 2003 Mar 7;278(10):8725-32. (PMID: 12486136); J Cell Biol. 2003 Mar 3;160(5):699-708. (PMID: 12604586); Nature. 2003 Dec 4;426(6966):563-6. (PMID: 14654841); Traffic. 2004 Feb;5(2):79-88. (PMID: 14690497); Cell Signal. 2004 Apr;16(4):401-13. (PMID: 14709330); Mol Biol Cell. 2004 Sep;15(9):4064-72. (PMID: 15254269); Methods Enzymol. 1974;30:368-87. (PMID: 4850210); EMBO J. 1986 Dec 1;5(12):3143-9. (PMID: 3816757); EMBO J. 1988 Apr;7(4):919-29. (PMID: 3402440); J Biol Chem. 1991 Apr 25;266(12):7910-8. (PMID: 1902231); J Cell Biol. 1993 Dec;123(6 Pt 1):1365-71. (PMID: 8253837); Cell. 1994 Sep 9;78(5):751-60. (PMID: 8087843); J Biol Chem. 1995 Jan 20;270(3):1337-41. (PMID: 7836400); J Biol Chem. 1995 Mar 3;270(9):4933-42. (PMID: 7876268); Nature. 1995 Dec 7;378(6557):632-5. (PMID: 8524399); Science. 1995 Dec 22;270(5244):1999-2002. (PMID: 8533093); EMBO J. 1996 Jun 3;15(11):2893-9. (PMID: 8654387); J Biol Chem. 1997 Feb 14;272(7):4141-8. (PMID: 9020126); EMBO J. 1997 Dec 15;16(24):7305-16. (PMID: 9405360); Proc Natl Acad Sci U S A. 1998 Mar 3;95(5):2140-5. (PMID: 9482852); Cell. 1998 Apr 17;93(2):263-75. (PMID: 9568718); Cell. 1998 May 1;93(3):423-32. (PMID: 9590176); Mol Biol Cell. 1998 Jun;9(6):1323-37. (PMID: 9614177); J Biol Chem. 1998 Sep 18;273(38):24786-91. (PMID: 9733781); Proc Natl Acad Sci U S A. 1998 Sep 15;95(19):11199-204. (PMID: 9736713); J Cell Sci. 1998 Oct;111 ( Pt 20):3081-90. (PMID: 9739081); J Cell Biol. 1998 Nov 16;143(4):947-55. (PMID: 9817753); Mol Cell. 1998 Nov;2(5):703-8. (PMID: 9844642); Cell. 1999 Feb 19;96(4):495-506. (PMID: 10052452)
Substance Nomenclature: 0 (Adaptor Protein Complex 2); 0 (Clathrin); 0 (GTPase-Activating Proteins); 0 (Liposomes); 0 (Protein Sorting Signals); 0 (Transcription Factor AP-1); 86-01-1 (Guanosine Triphosphate)
Entry Date(s): Date Created: 20050812 Date Completed: 20060214 Latest Revision: 20191210
Update Code: 20260130
PubMed Central ID: PMC1237080
DOI: 10.1091/mbc.e05-06-0568
PMID: 16093346
Database: MEDLINE

Journal Article; Research Support, Non-U.S. Gov't