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Familial concordance of breast cancer pathology as an indicator of genotype in multiple-case families

Title: Familial concordance of breast cancer pathology as an indicator of genotype in multiple-case families
Authors: Balleine, Rosemary L.; Provan, Pamela J.; Pupo, Gulietta M.; Pathmanathan, Nirmala; Cummings, Margaret; Farshid, Gelareh; Salisbury, Elizabeth L.; Bilous, A. Michael; Byth, Karen; Mann, Graham J.
Publisher Information: John Wiley & Sons
Publication Year: 2010
Collection: The University of Queensland: UQ eSpace
Subject Terms: Genome-wide association; Histopathological features; Estrogen-receptor; BRCA2 mutations; 1306 Cancer Research; 1311 Genetics
Description: The heterogeneity of multiple case breast cancer families that do not carry mutations in BRCA1 or BRCA2 (non-BRCA1/2 families) poses a challenge to the identification of breast cancer susceptibility genes. The aim of this study was to determine whether intrafamilial concordance in breast cancer pathology could identify subgroups of non-BRCA1/2 families with consistent genotypic features. Invasive breast cancers were reviewed from 84 individuals belonging to 30 multiple-case families; BRCA1 (n = 9), BRCA2 (n = 10), and non-BRCA1/2 (n = 11). Hierarchical cluster analysis based on histopathology and age at first diagnosis was then used to specify three subgroups designated Clusters 1-3. The genomic features of non-BRCA1/2 families were examined by genome wide linkage and FGFR2 SNP genotyping, according to whether they showed cluster-concordant or cluster-mixed familial pathology. The majority of pathogenic BRCA1 mutation carriers (80%) fell into a single cluster. In contrast pathogenic BRCA2 mutation carriers were distributed across all three clusters and within families, cluster groups were also generally mixed. Most non-BRCA1/2 mutation carriers belonged to Cluster 3 (71%). Genome wide linkage data from five non-BRCA1/2 Cluster 3-concordant families were compared with four mixed cluster non-BRCA1/2 families. This revealed a number of distinct linkage peaks, including some regions previously associated with breast cancer susceptibility. The distribution of low risk alleles in FGFR2 was not different between these two subgroups (P = 0.237). The pattern of breast cancer pathology concordance amongst family members may assist the investigation of breast cancer susceptibility in multiple case families.
Document Type: article in journal/newspaper
Language: English
ISSN: 1045-2257; 1098-2264
Availability: https://espace.library.uq.edu.au/view/UQ:219960
Accession Number: edsbas.1BC87438
Database: BASE