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Polyglutamine-expanded androgen receptor interferes with TFEB to elicit autophagy defects in SBMA

Title: Polyglutamine-expanded androgen receptor interferes with TFEB to elicit autophagy defects in SBMA
Authors: Cortes, Constanza J; Miranda, Helen C; Frankowski, Harald; Batlevi, Yakup; Young, Jessica E; Le, Amy; Ivanov, Nishi; Sopher, Bryce L; Carromeu, Cassiano; Muotri, Alysson R; Garden, Gwenn A; La Spada, Albert R
Source: Nature Neuroscience, vol 17, iss 9
Publisher Information: eScholarship, University of California
Publication Year: 2014
Collection: University of California: eScholarship
Subject Terms: 32 Biomedical and Clinical Sciences (for-2020); 3202 Clinical Sciences (for-2020); Brain Disorders (rcdc); Genetics (rcdc); Neurodegenerative (rcdc); Neurosciences (rcdc); Rare Diseases (rcdc); 2.1 Biological and endogenous factors (hrcs-rac); 1.1 Normal biological development and functioning (hrcs-rac); Neurological (hrcs-hc); Animals (mesh); Autophagy (mesh); Basic Helix-Loop-Helix Leucine Zipper Transcription Factors (mesh); Cellular Reprogramming (mesh); Disease Models; Animal (mesh); Female (mesh); Fibroblasts (mesh); Humans (mesh); Induced Pluripotent Stem Cells (mesh); Male (mesh); Mice; Transgenic (mesh); Motor Neurons (mesh); Muscular Disorders; Atrophic (mesh); Peptides (mesh); Phagosomes (mesh); Receptors; Androgen (mesh)
Time: 1180 - 1189
Description: Spinal and bulbar muscular atrophy (SBMA) is a neurodegenerative disorder that results from a polyglutamine repeat expansion in the androgen receptor (polyQ-AR). In this study, the authors show that autophagy is dysregulated in SBMA mice and in neural precursors obtained from iPSCs derived from human patients and that this results from an impaired interaction between AR and the transcription factor TFEB.
Document Type: article in journal/newspaper
File Description: application/pdf
Language: unknown
Relation: qt8v83h75n; https://escholarship.org/uc/item/8v83h75n; https://escholarship.org/content/qt8v83h75n/qt8v83h75n.pdf
DOI: 10.1038/nn.3787
Availability: https://escholarship.org/uc/item/8v83h75n; https://escholarship.org/content/qt8v83h75n/qt8v83h75n.pdf; https://doi.org/10.1038/nn.3787
Rights: public
Accession Number: edsbas.22432EBC
Database: BASE