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Interferon-β can induce progesterone receptors in human endometrial adenocarcinoma

Title: Interferon-β can induce progesterone receptors in human endometrial adenocarcinoma
Authors: Codegoni A. M.; Landoni F.; Lomonico S.; Losa G.; Mangioni C.; Taverna M.; Lucchini V.; D'Incalci M.
Contributors: Codegoni, A; Landoni, F; Lomonico, S; Losa, G; Mangioni, C; Taverna, M; Lucchini, V; D'Incalci, M
Publisher Information: WILEY
Publication Year: 1996
Collection: Università degli Studi di Milano-Bicocca: BOA (Bicocca Open Archive)
Subject Terms: endometrial adenocarcinoma; estrogen receptor; hIFN-β; progesterone receptor; Adenocarcinoma; Adult; Aged; 80 and over; Endometrial Neoplasm; Female; Human; Interferon-beta; Middle Aged; Receptors; Estrogen; Progesterone
Description: BACKGROUND. The induction of estrogen and progesterone receptors (ER and PGR) has been reported in breast and endometrial cancer cells exposed to human fibroblast interferon-β (hlFN-β). Clinical verification of this finding might provide the rationale for new therapeutic approaches. This study was designed to evaluate whether clinical treatment with high doses of hlFN-β induced ER and PGR in patients with endometrial adenocarcinoma. METHODS. Two biopsies were obtained, 1 before and 1 after hlFN-β treatment (3 x 106 i.m. every other day for 3 weeks) from 36 patients with endometrial adenocarcinoma. ER and PGR were determined with standard procedures using radiolabeled ligands. RESULTS. hIFN-β treatment did not affect the proportion of ER-positive (i.e., > 15 fmol/mg protein) or PGR-positive (i.e., >20 fmol/mg protein) cases. However, in patients with detectable ER and PGR at baseline, hIFN-β raised the levels. Using a 35% difference before and after therapy as a cut-off, 72 and 79% of cases had increases in ER and PGR, respectively. The difference was highly significant for PGR. CONCLUSiONS. In patients with endometrial adenocarcinoma with undetectable ER or PGR, hIFN- β did not induce the expression of these receptors. When the receptors were present they were upregulated by hIFN-β. Whether this increase in receptor levels, particularly PGR, has therapeutic applications remains to be established
Document Type: article in journal/newspaper
Language: English
Relation: info:eu-repo/semantics/altIdentifier/pmid/8697390; info:eu-repo/semantics/altIdentifier/wos/WOS:A1996UY25400011; volume:78; issue:3; firstpage:448; lastpage:453; numberofpages:6; journal:CANCER; https://hdl.handle.net/10281/265081
Availability: https://hdl.handle.net/10281/265081
Accession Number: edsbas.2528CF8
Database: BASE