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Murine Leukemia Virus Exploits Innate Sensing by Toll-Like Receptor 7 in B-1 Cells To Establish Infection and Locally Spread in Mice

Title: Murine Leukemia Virus Exploits Innate Sensing by Toll-Like Receptor 7 in B-1 Cells To Establish Infection and Locally Spread in Mice
Authors: Pi, Ruoxi; Iwasaki, Akiko; Sewald, Xaver; Mothes, Walther; Uchil, Pradeep D.
Contributors: Simon, Viviana; HHS | NIH | National Cancer Institute; HHS | NIH | National Institute of Allergy and Infectious Diseases; Howard Hughes Medical Institute
Source: Journal of Virology ; volume 93, issue 21 ; ISSN 0022-538X 1098-5514
Publisher Information: American Society for Microbiology
Publication Year: 2019
Description: Viruses establish infection in hosts by targeting highly permissive cell types. The retrovirus Friend murine leukemia virus (FrMLV) infects a subtype of B cells called B-1 cells that permit robust virus replication. The reason for their susceptibility had remained unknown. We found that innate sensing of incoming virus and the ensuing type I interferon response within B-1 cells are responsible for their observed susceptibility. Our data provide insights into how retroviruses coevolved with the host to co-opt innate immune sensing pathways designed to fight virus infections for establishing infection. Understanding early events in viral spread can inform antiviral intervention strategies that prevent the colonization of a host.
Document Type: article in journal/newspaper
Language: English
DOI: 10.1128/jvi.00930-19
DOI: 10.1128/JVI.00930-19
Availability: https://doi.org/10.1128/jvi.00930-19; https://journals.asm.org/doi/pdf/10.1128/JVI.00930-19
Rights: https://journals.asm.org/non-commercial-tdm-license
Accession Number: edsbas.28AC6012
Database: BASE