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Clathrin adaptor AP-1 and Stratum act in parallel pathways to control Notch activation in Drosophila Sensory Organ Precursor Cells

Title: Clathrin adaptor AP-1 and Stratum act in parallel pathways to control Notch activation in Drosophila Sensory Organ Precursor Cells
Authors: Bellec, Karen; Pinot, Mathieu; Gicquel, Isabelle; Le Borgne, Roland
Contributors: Institut de Génétique et Développement de Rennes (IGDR); Université de Rennes (UR)-Centre National de la Recherche Scientifique (CNRS)-Structure Fédérative de Recherche en Biologie et Santé de Rennes (Biosit : Biologie - Santé - Innovation Technologique); ANR-16-CE13-0004 Cytosign, Agence Nationale pour La Recherche; Fondation pour la Recherche Médiclae (FRM) FDT; ANR-16-CE13-0004,CYTOSIGN,Polarité épithéliale et signalisation au cours de la cytodiérèse des cellules animales(2016)
Source: ISSN: 0950-1991.
Publisher Information: HAL CCSD; Company of Biologists
Publication Year: 2021
Collection: Université de Rennes 1: Publications scientifiques (HAL)
Subject Terms: AP-1; Asymmetric cell division; Intracellular trafficking; Notch signaling; Stratum; [SDV]Life Sciences [q-bio]
Description: International audience ; Drosophila sensory organ precursors divide asymmetrically to generate pIIa/pIIb cells whose identity relies on activation of Notch at cytokinesis. While Notch is present apically and basally relative to the midbody at the pIIa-pIIb interface, the basal pool of Notch is reported to be the main contributor for Notch activation in the pIIa cell. Intra-lineage signaling requires appropriate apico-basal targeting of Notch, its ligand Delta and its trafficking partner Sanpodo. We previously reported that AP-1 and Stratum regulate the trafficking of Notch and Sanpodo from the trans-Golgi network to the basolateral membrane. Loss of AP-1 or Stratum caused mild Notch gain-of-function phenotypes. Here, we report that their concomitant loss results in a penetrant Notch gain-of-function phenotype indicating that they control parallel pathways. While unequal partitioning of cell fate determinants and cell polarity were unaffected, we observed increased amounts of signaling-competent Notch as well as Delta and Sanpodo at the apical pIIa-pIIb interface at the expense of the basal pool of Notch. We propose that AP-1 and Stratum operate in parallel pathways to localize Notch and control where receptor activation takes place.
Document Type: article in journal/newspaper
Language: English
Relation: info:eu-repo/semantics/altIdentifier/pmid/33298463; PUBMED: 33298463
DOI: 10.1242/dev.191437
Availability: https://hal.science/hal-03099273; https://hal.science/hal-03099273v1/document; https://hal.science/hal-03099273v1/file/dev.191437.full.pdf; https://doi.org/10.1242/dev.191437
Rights: http://creativecommons.org/licenses/by/ ; info:eu-repo/semantics/OpenAccess
Accession Number: edsbas.29E34A4D
Database: BASE