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Single-cell signaling network profiling during redox stress reveals dynamic redox regulation in immune cells

Title: Single-cell signaling network profiling during redox stress reveals dynamic redox regulation in immune cells
Authors: Yi-Chuan Wang; Ping-Hsun Wu; Wen-Chieh Ting; Yi-Fu Wang; Ming-Han Yang; Tung-Hung Su; Jia-Ying Su; Hsun-I Sun; Wei-Min Huang; Pei-Ling Tsai; Gerlinde Wernig; Ping-Chih Ho; Limei Wang; Chen-Tu Wu; Yih-Leong Chang; Tseng-Cheng Chen; Tzu-Ching Meng; Yao-Ming Chang; Shih-Lei Lai; Chia-Wei Li; Tai-Ming Ko; Kai-Chien Yang; Ya-Jen Chang; Yijuang Chern; Mei-Chuan Kuo; Yen-Tsung Huang; Yi-Shiuan Tzeng; Jih-Luh Tang; Shih-Yu Chen
Source: Nature Communications, Vol 16, Iss 1, Pp 1-18 (2025)
Publisher Information: Nature Portfolio
Publication Year: 2025
Collection: Directory of Open Access Journals: DOAJ Articles
Subject Terms: Science
Description: In eukaryotic cells, reactive oxygen species (ROS) serve as crucial signaling components. ROS are potentially toxic, so constant adjustments are needed to maintain cellular health. Here we describe a single-cell, mass cytometry-based method that we call signaling network under redox stress profiling (SN-ROP) to monitor dynamic changes in redox-related pathways during redox stress. SN-ROP quantifies ROS transporters, enzymes, oxidative stress products and associated signaling pathways to provide information on cellular redox regulation. Applied to diverse cell types and conditions, SN-ROP reveals unique redox patterns and dynamics including coordinated shifts in CD8+ T cells upon antigen stimulation as well as variations in CAR-T cell persistence. Furthermore, SN-ROP analysis uncovers environmental factors such as hypoxia and T cell exhaustion for influencing redox balance, and also reveals distinct features in patients on hemodialysis. Our findings thus support the use of SN-ROP to elucidate intricate redox networks and their implications in immune cell function and disease.
Document Type: article in journal/newspaper
Language: English
Relation: https://doi.org/10.1038/s41467-025-60727-z; https://doaj.org/toc/2041-1723; https://doaj.org/article/d3606999b8d1404fb58ca23cabc5bb7e
DOI: 10.1038/s41467-025-60727-z
Availability: https://doi.org/10.1038/s41467-025-60727-z; https://doaj.org/article/d3606999b8d1404fb58ca23cabc5bb7e
Accession Number: edsbas.2E07CE99
Database: BASE