Katalog Plus
Bibliothek der Frankfurt UAS
Bald neuer Katalog: sichern Sie sich schon vorab Ihre persönlichen Merklisten im Nutzerkonto: Anleitung.
Dieses Ergebnis aus BASE kann Gästen nicht angezeigt werden.  Login für vollen Zugriff.

UV-sensitive syndrome protein UVSSA recruits USP7 to regulate transcription-coupled repair

Title: UV-sensitive syndrome protein UVSSA recruits USP7 to regulate transcription-coupled repair
Authors: Schwertman, Petra; Lagarou, Anna; Dekkers, Dick; Raams, Anja; Hoek, Rianne; Laffeber, Charlie; Hoeijmakers, Jan; Demmers, Jeroen; Fousteri, M; Vermeulen, Wim; Marteijn, Jurgen
Source: Schwertman, P, Lagarou, A, Dekkers, D, Raams, A, Hoek, R, Laffeber, C, Hoeijmakers, J, Demmers, J, Fousteri, M, Vermeulen, W & Marteijn, J 2012, 'UV-sensitive syndrome protein UVSSA recruits USP7 to regulate transcription-coupled repair', Nature Genetics, vol. 44, no. 5, pp. 598-602. https://doi.org/10.1038/ng.2230
Publication Year: 2012
Subject Terms: /dk/atira/pure/keywords/researchprograms/AFL001000/EMCMGC011203; name=EMC MGC-01-12-03; /dk/atira/pure/keywords/researchprograms/AFL001000/EMCMGC022101; name=EMC MGC-02-21-01
Description: Transcription-coupled nucleotide-excision repair (TC-NER) is a subpathway of NER that efficiently removes the highly toxic RNA polymerase II blocking lesions in DNA. Defective TC-NER gives rise to the human disorders Cockayne syndrome and UV-sensitive syndrome (UVSS)(1). NER initiating factors are known to be regulated by ubiquitination(2). Using a SILAC-based proteomic approach, we identified UVSSA (formerly known as KIAA1530) as part of a UV-induced ubiquitinated protein complex. Knockdown of UVSSA resulted in TC-NER deficiency. UVSSA was found to be the causative gene for UVSS, an unresolved NER deficiency disorder(3). The UVSSA protein interacts with elongating RNA polymerase II, localizes specifically to UV-induced lesions, resides in chromatin-associated TC-NER complexes and is implicated in stabilizing the TC-NER master organizing protein ERCC6 (also known as CSB) by delivering the deubiquitinating enzyme USP7 to TC-NER complexes. Together, these findings indicate that UVSSA-USP7-mediated stabilization of ERCC6 represents a critical regulatory mechanism of TC-NER in restoring gene expression.
Document Type: article in journal/newspaper
Language: unknown
ISSN: 1061-4036; 1546-1718
Relation: info:eu-repo/semantics/altIdentifier/pmid/22466611; info:eu-repo/semantics/altIdentifier/pissn/1061-4036; info:eu-repo/semantics/altIdentifier/eissn/1546-1718
DOI: 10.1038/ng.2230
Availability: https://pure.eur.nl/en/publications/d1b6d9d3-495b-4755-b90b-383ef3ab918f; https://doi.org/10.1038/ng.2230; https://hdl.handle.net/1765/59964
Rights: info:eu-repo/semantics/closedAccess
Accession Number: edsbas.3D751D06
Database: BASE