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Post COVID‐19 syndrome with impairment of flow‐mediated epicardial vasodilation and flow reserve

Title: Post COVID‐19 syndrome with impairment of flow‐mediated epicardial vasodilation and flow reserve
Authors: Verma, Amanda; Ramayya, Tarun; Upadhyaya, Anand; Valenta, Ines; Lyons, Maureen; Marschall, Jonas; Dehdashti, Farrokh; Gropler, Robert J.; Woodard, Pamela K.; Schindler, Thomas Hellmut
Contributors: Washington University in St. Louis
Source: European Journal of Clinical Investigation ; volume 52, issue 12 ; ISSN 0014-2972 1365-2362
Publisher Information: Wiley
Publication Year: 2022
Collection: Wiley Online Library (Open Access Articles via Crossref)
Description: Aims The aim of this study is to evaluate whether post‐acute sequelae of COVID‐19 cardiovascular syndrome (PASC‐CVS) is associated with alterations in coronary circulatory function. Materials and Methods In individuals with PASC‐CVS but without known cardiovascular risk factors ( n = 23) and in healthy controls (CON, n = 23), myocardial blood flow (MBF) was assessed with 13 N‐ammonia and PET/CT in mL/g/min during regadenoson‐stimulated hyperemia, at rest, and the global myocardial flow reserve (MFR) was calculated. MBF was also measured in the mid and mid‐distal myocardium of the left ventricle (LV). The Δ longitudinal MBF gradient (hyperemia minus rest) as a reflection of an impairment of flow‐mediated epicardial vasodilation, was calculated. Results Resting MBF was significantly higher in PASC‐CVS than in CON (1.29 ± 0.27 vs. 1.08 ± 0.20 ml/g/min, p ≤ .024), while hyperemic MBFs did not differ significantly among groups (2.46 ± 0.53 and 2.40 ± 0.34 ml/g/min, p = .621). The MFR was significantly less in PASC‐CVS than in CON (1.97 ± 0.54 vs. 2.27 ± 0.43, p ≤ .031). In addition, there was a Δ longitudinal MBF gradient in PASC‐CVS, not observed in CON (−0.17 ± 0.18 vs. 0.04 ± 0.11 ml/g/min, p < .0001). Conclusions Post‐acute sequelae of COVID‐19 cardiovascular syndrome may be associated with an impairment of flow‐mediated epicardial vasodilation, while reductions in coronary vasodilator capacity appear predominantly related to increases in resting flow in women deserving further investigations.
Document Type: article in journal/newspaper
Language: English
DOI: 10.1111/eci.13871
Availability: https://doi.org/10.1111/eci.13871; https://onlinelibrary.wiley.com/doi/pdf/10.1111/eci.13871; https://onlinelibrary.wiley.com/doi/full-xml/10.1111/eci.13871
Rights: http://onlinelibrary.wiley.com/termsAndConditions#vor
Accession Number: edsbas.42F082F2
Database: BASE