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IL-22 driven neutrophils afford critical protection from acute murine cytomegalovirus infection (P6310)

Title: IL-22 driven neutrophils afford critical protection from acute murine cytomegalovirus infection (P6310)
Authors: Stacey, Maria; Marsden, Morgan; Dolton, Garry; Stack, Gabrielle; Jones, Emma; Taylor, Philip; Jones, Simon; Snelgrove, Robert; Wilkinson, Gavin; Humphreys, Ian
Source: The Journal of Immunology ; volume 190, issue Supplement_1, page 182.7-182.7 ; ISSN 0022-1767 1550-6606
Publisher Information: Oxford University Press (OUP)
Publication Year: 2013
Description: Pathogenic herpesviruses including cytomegalovirus target numerous host organs during infection, but the immunological mechanisms that afford antiviral protection in different tissue microenvironments require a better understanding. Utilizing the murine cytomegalovirus (MCMV) model, we demonstrate that neutrophils are critical antiviral effector cells that counter acute herpesvirus infection. Depletion of Ly6G+ neutrophils exacerbated virus-induced weight loss and elevated virus load in a tissue-restricted manner. Neutrophils profoundly and directly inhibited virus replication in vitro. MCMV-elicited neutrophil responses were orchestrated by interleukin-22 (IL-22), which was expressed by conventional NK cells and induced neutrophil-attractant chemokines in MCMV-infected peripheral organs. These data demonstrate that IL-22 affords anti-herpesvirus infection in peripheral tissue and highlights a previously unappreciated and critical role for neutrophils in countering cytomegalovirus infection.
Document Type: article in journal/newspaper
Language: English
DOI: 10.4049/jimmunol.190.supp.182.7
Availability: https://doi.org/10.4049/jimmunol.190.supp.182.7; https://academic.oup.com/jimmunol/article/190/Supplement_1/182.7/7999350
Rights: https://academic.oup.com/pages/standard-publication-reuse-rights
Accession Number: edsbas.535DA8AC
Database: BASE