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Notch Signaling Exacerbates Pulmonary Fibrosis by Regulating the Differentiation of CD4+ Tissue-Resident Memory T Cells

Title: Notch Signaling Exacerbates Pulmonary Fibrosis by Regulating the Differentiation of CD4+ Tissue-Resident Memory T Cells
Authors: Jia Shi; Ruiting Su; Lili Zhuang; Zhangmei Lin; Xinyuan Ruan; Yichao Qian; Jieying Zhu; Shuyi Wang; Niansheng Yang
Source: Biomolecules ; Volume 16 ; Issue 2 ; Pages: 328
Publisher Information: Multidisciplinary Digital Publishing Institute
Publication Year: 2026
Collection: MDPI Open Access Publishing
Subject Terms: pulmonary fibrosis; CD4 + tissue resident memory T cells; Notch signaling
Description: The involvement of the immune system in pulmonary fibrosis is established, the precise contributions of tissue-resident memory T (TRM) cells are still poorly defined. This study sought to define the contribution of CD4+ TRM cells to pulmonary fibrosis, their origin, and regulatory mechanisms. We combined bioinformatic analysis of human fibrotic lung single-cell RNA-sequencing data with experiments in a bleomycin-induced C57BL/6 mouse model. Flow cytometry, targeted in vivo depletion, lymphocyte trafficking blockade, cell co-culture, and pharmacological inhibition were employed. CD4+ TRM cells were observed at higher frequencies within fibrotic lung tissue. Their presence correlated with disease severity, and they were found to exhibit a pro-inflammatory and pro-fibrotic phenotype. Their specific depletion alleviated fibrosis. These cells primarily originated from recruited circulating lymphocytes, as blocking this recruitment reduced TRM accumulation and attenuated disease. Furthermore, the Notch signaling pathway was activated in fibrotic lung CD4+ TRM cells, and its inhibition suppressed their differentiation and impaired their pro-fibrotic function. We conclude that CD4+ TRM cells are pathogenic drivers in pulmonary fibrosis, originating from circulating precursors and being regulated by Notch signaling, underscoring their relevance for therapeutic intervention.
Document Type: text
File Description: application/pdf
Language: English
Relation: Cellular Biochemistry; https://dx.doi.org/10.3390/biom16020328
DOI: 10.3390/biom16020328
Availability: https://doi.org/10.3390/biom16020328
Rights: https://creativecommons.org/licenses/by/4.0/
Accession Number: edsbas.569B7FB4
Database: BASE