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The alanine-serine-cysteine-1 (Asc-1) transporter controls glycine levels in the brain and is required for glycinergic inhibitory transmission

Title: The alanine-serine-cysteine-1 (Asc-1) transporter controls glycine levels in the brain and is required for glycinergic inhibitory transmission
Authors: Safory, Hazem; Neame, Samah; Shulman, Yoav; Zubedat, Salman; Radzishevsky, Inna; Rosenberg, Dina; Sason, Hagit; Engelender, Simone; Avital, Avi; Huelsmann, Swen; Schiller, Jackie; Wolosker, Herman
Contributors: Safory, Hazem; Neame, Samah; Shulman, Yoav; Zubedat, Salman; Radzishevsky, Inna; Rosenberg, Dina; Sason, Hagit; Engelender, Simone; Avital, Avi; Huelsmann, Swen; Schiller, Jackie; Wolosker, Herman
Publisher Information: Wiley-blackwell
Publication Year: 2015
Collection: Georg-August-Universität Göttingen: GoeScholar
Description: Asc-1 (SLC7A10) is an amino acid transporter whose deletion causes neurological abnormalities and early postnatal death in mice. Using metabolomics and behavioral and electrophysiological methods, we demonstrate that Asc-1 knockout mice display a marked decrease in glycine levels in the brain and spinal cord along with impairment of glycinergic inhibitory transmission, and a hyperekplexia-like phenotype that is rescued by replenishing brain glycine. Asc-1 works as a glycine and L-serine transporter, and its transport activity is required for the subsequent conversion of L-serine into glycine invivo. Asc-1 is a novel regulator of glycine metabolism and a candidate for hyperekplexia disorders.
Document Type: article in journal/newspaper
Language: unknown
Relation: 000354131600011
DOI: 10.15252/embr.201439561
Availability: https://resolver.sub.uni-goettingen.de/purl?gro-2/37246; https://doi.org/10.15252/embr.201439561
Rights: info:eu-repo/semantics/openAccess
Accession Number: edsbas.6A770366
Database: BASE