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Bradykinin Contributes to the Systemic Hemodynamic Effects of Chronic Angiotensin-Converting Enzyme Inhibition in Patients With Heart Failure

Title: Bradykinin Contributes to the Systemic Hemodynamic Effects of Chronic Angiotensin-Converting Enzyme Inhibition in Patients With Heart Failure
Authors: Cruden, Nicholas L.M.; Witherow, Fraser N.; Webb, David J.; Fox, Keith A.A.; Newby, David E.
Source: Arteriosclerosis, Thrombosis, and Vascular Biology ; volume 24, issue 6, page 1043-1048 ; ISSN 1079-5642 1524-4636
Publisher Information: Ovid Technologies (Wolters Kluwer Health)
Publication Year: 2004
Description: Background— Bradykinin is an endogenous vasodilator that may contribute to the systemic effects of angiotensin-converting enzyme (ACE) inhibitor therapy. Using B9340, a bradykinin receptor antagonist, we determined the contribution of bradykinin to the systemic hemodynamic effects of long-term ACE inhibition in patients with chronic heart failure. Methods and Results— Fourteen patients with heart failure received enalapril (10 mg twice daily) or losartan (50 mg twice daily) in a randomized double-blind crossover trial. After 6 weeks treatment, patients underwent right heart catheterization and were randomized to an intravenous infusion of B9340 (2 to 20 μg/kg per minute) or saline placebo. After B9340 infusion in patients treated with enalapril, mean arterial pressure (+5.2 mm Hg), systemic vascular resistance (+315 dynes·s/cm 5 ), pulmonary arterial wedge pressure (−1.4 mm Hg), and mean pulmonary arterial pressure (−1.3 mm Hg) were greater compared with losartan ( P
Document Type: article in journal/newspaper
Language: English
DOI: 10.1161/01.atv.0000129331.21092.1d
DOI: 10.1161/01.ATV.0000129331.21092.1d
Availability: https://doi.org/10.1161/01.atv.0000129331.21092.1d; https://www.ahajournals.org/doi/full/10.1161/01.ATV.0000129331.21092.1d
Accession Number: edsbas.7EF5A77A
Database: BASE