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Pharmacological inhibition of frizzled 4 delays cell cycle progression and limits oral squamous cell carcinoma growth

Title: Pharmacological inhibition of frizzled 4 delays cell cycle progression and limits oral squamous cell carcinoma growth
Authors: Destefani, Riccardo; Valookkaran, Ilaria J.; Bensland, Sabrina; Meisel, Christian T.; Porcheri, Cristina
Contributors: Olga Mayenfisch Stiftung; Julius Müller Stiftung; Krebsliga Schweiz; Julius Klaus-Stiftung für Genetik und Sozialanthropologie
Source: Frontiers in Cell and Developmental Biology ; volume 14 ; ISSN 2296-634X
Publisher Information: Frontiers Media SA
Publication Year: 2026
Collection: Frontiers (Publisher - via CrossRef)
Description: Oral squamous cell carcinoma is an aggressive malignancy driven by aberrant signaling pathways, with Wnt signaling acting as a central regulator of proliferation, tumor-microenvironment interactions, and oncogenic growth. Here, we focus on Frizzled-4, a Wnt receptor with context-dependent functions in epithelial cancers. We show that pharmacological inhibition of Frizzled-4 delays cell cycle progression, and reduces proliferative capacity. These effects are accompanied by suppressed metabolism of retinoic acid, suggesting that Frizzled-4 inhibition stabilizes intracellular retinoic acid levels, which ultimately contributes to the observed cell cycle slowdown. Blocking retinoic acid signaling reverses the cell cycle effects of Frizzled-4 inhibition, confirming that retinoic acid-dependent regulation operates downstream of Frizzled-4. By restraining uncontrolled proliferation and attenuating oncogenic signaling, Frizzled-4 emerges as a key molecular node in oral squamous cell carcinoma, highlighting its potential as a therapeutic target to counteract Wnt-driven tumor growth.
Document Type: article in journal/newspaper
Language: unknown
DOI: 10.3389/fcell.2026.1756565
DOI: 10.3389/fcell.2026.1756565/full
Availability: https://doi.org/10.3389/fcell.2026.1756565; https://www.frontiersin.org/articles/10.3389/fcell.2026.1756565/full
Rights: https://creativecommons.org/licenses/by/4.0/
Accession Number: edsbas.995DA5EF
Database: BASE