Katalog Plus
Bibliothek der Frankfurt UAS
Bald neuer Katalog: sichern Sie sich schon vorab Ihre persönlichen Merklisten im Nutzerkonto: Anleitung.
Dieses Ergebnis aus BASE kann Gästen nicht angezeigt werden.  Login für vollen Zugriff.

Mapping Attenuation Determinants in Enterovirus-D68

Title: Mapping Attenuation Determinants in Enterovirus-D68
Authors: Yeh, Ming Te; Capponi, Sara; Catching, Adam; Bianco, Simone; Andino, Raul
Source: Viruses, vol 12, iss 8
Publisher Information: eScholarship, University of California
Publication Year: 2020
Collection: University of California: eScholarship
Subject Terms: Microbiology; Biological Sciences; Neurosciences; Biodefense; Infectious Diseases; Genetics; Rare Diseases; Emerging Infectious Diseases; 2.1 Biological and endogenous factors; 2.2 Factors relating to the physical environment; Infection; Good Health and Well Being; 5' Untranslated Regions; Amino Acid Substitution; Animals; Brain; Capsid Proteins; Cell Line; Tumor; Disease Models; Animal; Enterovirus D; Human; Enterovirus Infections; Genes; Viral; Humans; Mice; Inbred C57BL; Knockout
Description: Enterovirus (EV)-D68 has been associated with epidemics in the United Sates in 2014, 2016 and 2018. This study aims to identify potential viral virulence determinants. We found that neonatal type I interferon receptor knockout mice are susceptible to EV-D68 infection via intraperitoneal inoculation and were able to recapitulate the paralysis process observed in human disease. Among the EV-D68 strains tested, strain US/MO-14-18949 caused no observable disease in this mouse model, whereas the other strains caused paralysis and death. Sequence analysis revealed several conserved genetic changes among these virus strains: nucleotide positions 107 and 648 in the 5'-untranslated region (UTR); amino acid position 88 in VP3; 1, 148, 282 and 283 in VP1; 22 in 2A; 47 in 3A. A series of chimeric and point-mutated infectious clones were constructed to identify viral elements responsible for the distinct virulence. A single amino acid change from isoleucine to valine at position 88 in VP3 attenuated neurovirulence by reducing virus replication in the brain and spinal cord of infected mice.
Document Type: article in journal/newspaper
File Description: application/pdf
Language: unknown
Relation: qt3wn5714m; https://escholarship.org/uc/item/3wn5714m; https://escholarship.org/content/qt3wn5714m/qt3wn5714m.pdf
DOI: 10.3390/v12080867
Availability: https://escholarship.org/uc/item/3wn5714m; https://escholarship.org/content/qt3wn5714m/qt3wn5714m.pdf; https://doi.org/10.3390/v12080867
Rights: CC-BY
Accession Number: edsbas.9B3824A9
Database: BASE