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Phenome-wide functional dissection of pleiotropic effects highlights key molecular pathways for human complex traits

Title: Phenome-wide functional dissection of pleiotropic effects highlights key molecular pathways for human complex traits
Authors: Shikov, AE; Skitchenko, RK; Predeus, AV; Barbitoff, YA
Publisher Information: Springer Science and Business Media LLC
Publication Year: 2020
Collection: The University of Liverpool Repository
Description: Over the recent decades, genome-wide association studies (GWAS) have dramatically changed the understanding of human genetics. A recent genetic data release by UK Biobank (UKB) has allowed many researchers worldwide to have comprehensive look into the genetic architecture of thousands of human phenotypes. In this study, we used GWAS summary statistics derived from the UKB cohort to investigate functional mechanisms of pleiotropic effects across the human phenome. We find that highly pleiotropic variants often correspond to broadly expressed genes with ubiquitous functions, such as matrisome components and cell growth regulators; and tend to colocalize with tissue-shared eQTLs. At the same time, signaling pathway components are more prevalent among highly pleiotropic genes compared to regulatory proteins such as transcription factors. Our results suggest that protein-level pleiotropy mediated by ubiquitously expressed genes is the most prevalent mechanism of pleiotropic genetic effects across the human phenome.
Document Type: article in journal/newspaper
Language: English
ISSN: 2045-2322
Relation: Shikov, AE, Skitchenko, RK orcid:0000-0002-7203-8206 , Predeus, AV orcid:0000-0002-2750-1599 and Barbitoff, YA (2020) Phenome-wide functional dissection of pleiotropic effects highlights key molecular pathways for human complex traits Scientific Reports, 10 (1). 1037-. ISSN 2045-2322, 2045-2322
DOI: 10.1038/s41598-020-58040-4
Availability: https://livrepository.liverpool.ac.uk/3075709/; https://doi.org/10.1038/s41598-020-58040-4
Accession Number: edsbas.A4D53278
Database: BASE