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Interleukin-38 is a negative regulator of trained immunity—A retrospective multi-omics study

Title: Interleukin-38 is a negative regulator of trained immunity—A retrospective multi-omics study
Authors: Teufel, LU; Matzaraki, V; Folkman, L; de Graaf, DM; Horst, RT; Moorlag, SJCFM; dos Santos, JC; Mulders-Manders, CM; Krausgruber, T; Dinarello, C; Netea, MG; Joosten, LAB; Arts, RJW
Publisher Information: Elsevier
Publication Year: 2025
Collection: Griffith University: Griffith Research Online
Subject Terms: Immunology; Biomedical and clinical sciences
Description: Trained immunity is a long-lasting innate immune cell phenotype with benefits in infection control and recognized anti-cancer effects. Conversely, inappropriately induced trained immunity contributes to pathological inflammation, warranting the exploration of regulatory pathways. We explore interleukin-38 (IL-38) as a regulator of trained immunity in vivo in a cohort of 325 healthy adults vaccinated with Bacillus Calmette-Guérin (BCG). Using multi-omics profiling, we find that IL-38 is negatively associated with trained immunity on metabolic and epigenetic level. Genetic variants in IL1F10, encoding for IL-38, further link IL-38 to diminished training responses. These associations were validated in human and murine models. We confirmed that IL-38 functionally impairs anti-microbial traits of trained immunity in trained immunity-infection models in vivo (IL-38KO mice) and in vitro (human monocytes). Our study therefore suggests that IL-38 endogenously regulates the induction of trained immunity in humans in vivo. ; Full Text
Document Type: article in journal/newspaper
Language: English
Relation: iScience; Teufel, LU; Matzaraki, V; Folkman, L; de Graaf, DM; Horst, RT; Moorlag, SJCFM; dos Santos, JC; Mulders-Manders, CM; Krausgruber, T; Dinarello, C; Netea, MG; Joosten, LAB; Arts, RJW, Interleukin-38 is a negative regulator of trained immunity—A retrospective multi-omics study, iScience, 2025, 28 (11), pp. 113758; https://hdl.handle.net/10072/440189
DOI: 10.1016/j.isci.2025.113758
Availability: https://hdl.handle.net/10072/440189; https://doi.org/10.1016/j.isci.2025.113758
Rights: https://creativecommons.org/licenses/by/4.0/ ; © 2025 The Author(s). Published by Elsevier Inc. This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/). ; open access
Accession Number: edsbas.AAF05761
Database: BASE