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Obesity and overweight are linked to increased sodium-glucose cotransporter 1 and glucose transporter 5 levels in duodenum

Title: Obesity and overweight are linked to increased sodium-glucose cotransporter 1 and glucose transporter 5 levels in duodenum
Authors: Fiorentino T. V.; De Vito F.; Suraci E.; Marasco R.; Hribal M. L.; Luzza F.; Sesti G.
Contributors: Fiorentino, T. V.; De Vito, F.; Suraci, E.; Marasco, R.; Hribal, M. L.; Luzza, F.; Sesti, G.
Publication Year: 2023
Collection: Sapienza Università di Roma: CINECA IRIS
Subject Terms: Obesity; overweight; sodium-glucose cotransporter 1; duodenum
Description: Objective: Prior evidence indicates that individuals with obesity have an accelerated intestinal glucose absorption. This cross-sectional study evaluated whether those with overweight or obesity display higher duodenal protein levels of the glucose carriers sodium-glucose cotransporter 1 (SGLT-1), glucose transporter 2 (GLUT-2), and glucose transporter 5 (GLUT-5). Methods: SGLT-1, GLUT-2, and GLUT-5 protein levels were assessed on duodenal mucosa biopsies of 52 individuals without diabetes categorized on the basis of their BMI as lean, with overweight, or with obesity. Results: Individuals with overweight and obesity exhibited progressively increased duodenal protein levels of SGLT-1 and GLUT-5 as compared with the lean group. Conversely, no differences in duodenal GLUT-2 abundance were found among the three groups. Univariate analysis showed that SGLT-1 and GLUT-5 protein levels were positively correlated with BMI, waist circumference, 1-hour post-load glucose, fasting and post-load insulin, and insulin secretion and resistance levels. Furthermore, a positive relationship was detected between intestinal GLUT-5 levels and serum uric acid concentrations, a product of fructose metabolism known to be involved in the pathogenesis of obesity and its complications. Conclusions: Individuals with overweight and obesity display enhanced duodenal SGLT-1 and GLUT-5 abundance, which correlates with increased postprandial glucose concentrations, insulin resistance, and hyperinsulinemia.
Document Type: article in journal/newspaper
Language: English
Relation: info:eu-repo/semantics/altIdentifier/pmid/36746764; info:eu-repo/semantics/altIdentifier/wos/WOS:000928807900001; volume:31; issue:3; firstpage:724; lastpage:731; numberofpages:8; journal:OBESITY; https://hdl.handle.net/11573/1671233
DOI: 10.1002/oby.23653
Availability: https://hdl.handle.net/11573/1671233; https://doi.org/10.1002/oby.23653
Rights: info:eu-repo/semantics/closedAccess ; license:Tutti i diritti riservati (All rights reserved) ; license uri:iris.PRI00
Accession Number: edsbas.AFE94D5F
Database: BASE