Katalog Plus
Bibliothek der Frankfurt UAS
Bald neuer Katalog: sichern Sie sich schon vorab Ihre persönlichen Merklisten im Nutzerkonto: Anleitung.
Dieses Ergebnis aus BASE kann Gästen nicht angezeigt werden.  Login für vollen Zugriff.

Omega‐3 fatty acid supplementation does not attenuate declines in skeletal muscle mitochondrial area in young, healthy females during immobilization

Title: Omega‐3 fatty acid supplementation does not attenuate declines in skeletal muscle mitochondrial area in young, healthy females during immobilization
Authors: Megan M. Lo; Merryl N. Black; Chris McGlory; Ravninder Bahniwal; Michael Kamal; Joe Quadrilatero; Stuart M. Phillips; Michaela C. Devries
Source: Physiological Reports, Vol 14, Iss 2, Pp n/a-n/a (2026)
Publisher Information: Wiley
Publication Year: 2026
Collection: Directory of Open Access Journals: DOAJ Articles
Subject Terms: electron microscopy; mitochondria; muscle disuse; omega‐3 fatty acid; skeletal muscle; Physiology; QP1-981
Description: Mitochondrial subcellular area influences function. Muscle disuse reduces mitochondrial content; however, its effect on mitochondrial subcellular location is unclear. Omega‐3 fatty acid (n‐3) attenuates declines in muscle mass and mitochondrial function during disuse; however, whether n‐3 supplementation prevents the decline in mitochondrial content has not been examined. We investigated the effects of 2 weeks of leg immobilization followed by 2 weeks of remobilization on skeletal muscle mitochondrial content and subcellular localization with and without n‐3 supplementation. Twenty healthy females supplemented with n‐3 (2.97 g EPA and 2.03 g DHA) or control (isoenergetic sunflower oil) during 2 weeks of unilateral leg immobilization and 2 weeks of remobilization. Vastus lateralis biopsies were taken for electron microscopic analysis of mitochondrial content. Subsarcolemmal (SS) mitochondrial content decreased during immobilization (control: −9%, n‐3: −66%, p = 0.009) and remained lower following recovery (control: −41%, n‐3: −42%, p = 0.005). This effect was driven by the n‐3 group (p < 0.02). Intermyofibrillar (IMF) mitochondrial content did not decline during immobilization, but was lower than baseline following recovery in the central (p=0.01) IMF. The effects of leg immobilization on mitochondrial content differ by location, are not reversed with short‐term recovery, and are influenced by n‐3 supplementation.
Document Type: article in journal/newspaper
Language: English
Relation: https://doi.org/10.14814/phy2.70736; https://doaj.org/toc/2051-817X; https://doaj.org/article/b99902945da846e6a2dcd19dcb0a426f
DOI: 10.14814/phy2.70736
Availability: https://doi.org/10.14814/phy2.70736; https://doaj.org/article/b99902945da846e6a2dcd19dcb0a426f
Accession Number: edsbas.B0158138
Database: BASE