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Specific cancer-associated mutations in the switch III region of Ras increase tumorigenicity by nanocluster augmentation

Title: Specific cancer-associated mutations in the switch III region of Ras increase tumorigenicity by nanocluster augmentation
Authors: Solman, Maja; Ligabue, Alessio; Blazevits, Olga; Jaiswal, Alok; Zhou, Yong; Liang, Hong; Lectez, Benoit; Kopra, Kari; Guzman, Camilo; Harma, Harri; Hancock, John F.; Aittokallio, Tero; Abankwa, Daniel
Contributors: Institute for Molecular Medicine Finland; Tero Aittokallio / Principal Investigator; Bioinformatics
Publisher Information: ELIFE SCIENCES PUBLICATIONS LTD
Publication Year: 2016
Collection: Helsingfors Universitet: HELDA – Helsingin yliopiston digitaalinen arkisto
Subject Terms: H-RAS; K-RAS; PLASMA-MEMBRANE; DEVELOPMENTAL DISORDERS; QUANTITATIVE-ANALYSIS; SIGNAL-TRANSDUCTION; PROTEIN-KINASE; FORMS DIMERS; NUCLEOTIDE; ORIENTATION; Biomedicine; Cancers
Description: Hotspot mutations of Ras drive cell transformation and tumorigenesis. Less frequent mutations in Ras are poorly characterized for their oncogenic potential. Yet insight into their mechanism of action may point to novel opportunities to target Ras. Here, we show that several cancer-associated mutations in the switch III region moderately increase Ras activity in all isoforms. Mutants are biochemically inconspicuous, while their clustering into nanoscale signaling complexes on the plasma membrane, termed nanocluster, is augmented. Nanoclustering dictates downstream effector recruitment, MAPK-activity, and tumorigenic cell proliferation. Our results describe an unprecedented mechanism of signaling protein activation in cancer. ; Peer reviewed
Document Type: article in journal/newspaper
File Description: application/pdf
Language: English
ISBN: 978-84-941364-4-3; 84-941364-4-5
Relation: Suomen Akatemia (Academy of Finland) 272437, 269862, 279163 Tero Aittokallio; https://hdl.handle.net/10138/161430; 84941364451; 000373812900001
Availability: https://hdl.handle.net/10138/161430
Rights: info:eu-repo/semantics/openAccess ; openAccess
Accession Number: edsbas.BA6C17D2
Database: BASE