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Biogenesis of Extracellular Vesicles during Herpes Simplex Virus 1 Infection: Role of the CD63 Tetraspanin

Title: Biogenesis of Extracellular Vesicles during Herpes Simplex Virus 1 Infection: Role of the CD63 Tetraspanin
Authors: Dogrammatzis, Christos; Deschamps, Thibaut; Kalamvoki, Maria
Contributors: Sandri-Goldin, Rozanne M.; HHS | NIH | NIH Office of the Director; KU | University of Kansas Medical Center
Source: Journal of Virology ; volume 93, issue 2 ; ISSN 0022-538X 1098-5514
Publisher Information: American Society for Microbiology
Publication Year: 2019
Description: Intercellular communication, especially in neurons, largely relies on EVs, and modulation of EVs is known to impact physiological processes. Here, we present evidence that HSV-1 infection causes major alterations in the biogenesis of EVs, including an increase in their number and an increase in the CD63-positive population of EVs. These alterations result in an enrichment of the milieu of infection with EVs carrying signatures from infected cells. In addition to changes in the origin and type, EVs released by infected cells have differences in cargo, as they carry viral and host factors determined by the virus. The tetraspanin CD63 negatively impacts the infection, as demonstrated by CD63-knockdown and overexpression assays. A proposed mechanism involves the activation of antiviral responses in cells receiving CD63-positive EVs released by infected cells. Overall, HSV-1 causes major alterations in EVs that could contribute to HSV-1 persistence and pathogenesis.
Document Type: article in journal/newspaper
Language: English
DOI: 10.1128/jvi.01850-18
DOI: 10.1128/JVI.01850-18
Availability: https://doi.org/10.1128/jvi.01850-18; https://journals.asm.org/doi/pdf/10.1128/JVI.01850-18
Rights: https://journals.asm.org/non-commercial-tdm-license
Accession Number: edsbas.BDC07D35
Database: BASE