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Systemic and skin-limited delayed-type drug hypersensitivity reactions associate with distinct resident and recruited T cell subsets

Title: Systemic and skin-limited delayed-type drug hypersensitivity reactions associate with distinct resident and recruited T cell subsets
Authors: Shah, Pranali N; Romar, George A; Manukyan, Artür; Ko, Wei-Che; Hsieh, Pei-Chen; Velasquez, Gustavo A; Schunkert, Elisa M; Fu, Xiaopeng; Guleria, Indira; Bronson, Roderick T; Wei, Kevin; Waldman, Abigail H; Vleugels, Frank R; Liang, Marilyn G; Giobbie-Hurder, Anita; Mostaghimi, Arash; Schmidt, Birgitta Ar; Barrera, Victor; Foreman, Ruth K; Garber, Manuel; Divito, Sherrie J
Contributors: Dermatology; Genomics and Computational Biology; Garber Lab
Source: The Journal of clinical investigation ; United States
Publication Year: 2024
Collection: University of Massachusetts, Medical School: eScholarship@UMMS
Subject Terms: Allergy; Dermatology; Immunology; Skin; T cells
Description: Delayed-type drug hypersensitivity reactions are major causes of morbidity and mortality. The origin, phenotype and function of pathogenic T cells across the spectrum of severity requires investigation. We leveraged recent technical advancements to study skin-resident memory T cells (TRM) versus recruited T cell subsets in the pathogenesis of severe systemic forms of disease, SJS/TEN and DRESS, and skin-limited disease, morbilliform drug eruption (MDE). Microscopy, bulk transcriptional profiling and scRNAseq + CITEseq + TCRseq supported in SJS/TEN clonal expansion and recruitment of cytotoxic CD8+ T cells from circulation into skin, along with expanded and non-expanded cytotoxic CD8+ skin TRM. Comparatively, MDE displayed a cytotoxic T cell profile in skin without appreciable expansion and recruitment of cytotoxic CD8+ T cells from circulation, implicating TRM as potential protagonists in skin-limited disease. Mechanistic interrogation in patients unable to recruit T cells from circulation into skin and in a parallel mouse model supported that skin TRM were sufficient to mediate MDE. Concomitantly, SJS/TEN displayed a reduced regulatory T cell (Treg) signature compared to MDE. DRESS demonstrated recruitment of cytotoxic CD8+ T cells into skin like SJS/TEN, yet a pro-Treg signature like MDE. These findings have important implications for fundamental skin immunology and clinical care.
Document Type: text
File Description: application/pdf
Language: English
Relation: Journal of Clinical Investigation; https://doi.org/10.1172/jci178253; http://hdl.handle.net/20.500.14038/53742; The Journal of clinical investigation
DOI: 10.1172/JCI178253
Availability: https://doi.org/10.1172/JCI178253; https://hdl.handle.net/20.500.14038/53742
Rights: Copyright © 2024, Shah et al. This work is licensed under the Creative Commons Attribution 4.0 International License. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/. ; https://creativecommons.org/licenses/by/4.0/
Accession Number: edsbas.C3BD544C
Database: BASE