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Prediction of a New Ligand-Binding Site for Type 2 Motif based on the Crystal Structure of ALG-2 by Dry and Wet Approaches

Title: Prediction of a New Ligand-Binding Site for Type 2 Motif based on the Crystal Structure of ALG-2 by Dry and Wet Approaches
Authors: Masatoshi Maki; Takeshi Takahashi; Hironori Suzuki; Hideki Shibata; Tatsutoshi Inuzuka
Source: International Journal of Molecular Sciences, Vol 13, Iss 6, Pp 7532-7549 (2012)
Publisher Information: MDPI AG
Publication Year: 2012
Collection: Directory of Open Access Journals: DOAJ Articles
Subject Terms: ALG-2; calcium-binding protein; computational prediction; protein-protein interaction; proline-rich motif; Biology (General); QH301-705.5; Chemistry; QD1-999
Description: ALG-2 is a penta-EF-hand Ca 2+ -binding protein and interacts with a variety of intracellular proteins. Two types of ALG-2-binding motifs have been determined: type 1, P X YP X nYP ( X , variable; n = 4), in ALIX and PLSCR3; type 2, P X PGF, in Sec31A and PLSCR3. The previously solved X-ray crystal structure of the complex between ALG-2 and an ALIX peptide containing type 1 motif showed that the peptide binds to Pocket 1 and Pocket 2. Co-crystallization of ALG-2 and type 2 motif-containing peptides has not been successful. To gain insights into the molecular basis of type 2 motif recognition, we searched for a new hydrophobic cavity by computational algorithms using MetaPocket 2.0 based on 3D structures of ALG-2. The predicted hydrophobic pocket designated Pocket 3 fits with N -acetyl-ProAlaProGlyPhe-amide, a virtual penta-peptide derived from one of the two types of ALG-2-binding sites in PLSCR3 (type 2 motif), using the molecular docking software AutoDock Vina. We investigated effects of amino acid substitutions of the predicted binding sites on binding abilities by pulldown assays using glutathione- S -transferase -fused ALG-2 of wild-type and mutant proteins and lysates of cells expressing green fluorescent protein -fused PLSCR3 of wild-type and mutants. Substitution of either L52 with Ala or F148 with Ser of ALG-2 caused loss of binding abilities to PLSCR3 lacking type 1 motif but retained those to PLSCR3 lacking type 2 motif, strongly supporting the hypothesis that Pocket 3 is the binding site for type 2 motif.
Document Type: article in journal/newspaper
Language: English
Relation: http://www.mdpi.com/1422-0067/13/6/7532; https://doaj.org/toc/1422-0067; https://doaj.org/article/dd2be9a6344c4666ae6a01956fe05346
DOI: 10.3390/ijms13067532
Availability: https://doi.org/10.3390/ijms13067532; https://doaj.org/article/dd2be9a6344c4666ae6a01956fe05346
Accession Number: edsbas.CC09D080
Database: BASE