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The adaptor protein SKT interacts with PSD-95 and SHANK3 and affects synaptic functions

Title: The adaptor protein SKT interacts with PSD-95 and SHANK3 and affects synaptic functions
Authors: Morellato A.; De Gregorio M.; Angelini C.; Torelli F.; Belmonte V.; Alfieri A.; Bersia B.; Cravero T.; Gai M.; Bianciotto O. T.; Oddone R.; Salemme V.; Natalini D.; Centonze G.; Nigrelli F.; Raspanti A.; Gurgone A.; D'Attanasio G.; Mele P.; Eva C.; Lodi A.; Gavello D.; Carabelli V.; Hidisoglu E.; Chiantia G.; Carbone E.; Becchetti A.; Giustetto M.; Marcantoni A.; Turco E.; Bertocchi I.; Defilippi P.
Contributors: Morellato, A; De Gregorio, M; Angelini, C; Torelli, F; Belmonte, V; Alfieri, A; Bersia, B; Cravero, T; Gai, M; Bianciotto, O; Oddone, R; Salemme, V; Natalini, D; Centonze, G; Nigrelli, F; Raspanti, A; Gurgone, A; D'Attanasio, G; Mele, P; Eva, C; Lodi, A; Gavello, D; Carabelli, V; Hidisoglu, E; Chiantia, G; Carbone, E; Becchetti, A; Giustetto, M; Marcantoni, A; Turco, E; Bertocchi, I; Defilippi, P
Publisher Information: Cell Press; NL
Publication Year: 2025
Collection: Università degli Studi di Milano-Bicocca: BOA (Bicocca Open Archive)
Subject Terms: behavior; CP: Neuroscience; electrophysiology; KIAA1217; postsynaptic density; PSD-95; SHANK3; SKT; spine morphology; synaptic plasticity; Settore BIOS-06/A - Fisiologia
Description: Postsynaptic density (PSD) is a tightly interconnected protein network ensuring synaptic function through the interaction of neurotransmitter receptors, structural adaptor proteins, and signaling molecules. Disruption of PSD may cause neurological diseases, including autism spectrum disorders and cognitive impairment. We demonstrate that the SKT adaptor distinctly localizes within dendritic spines as an integral component of the synaptic network, binding PSD-95 and SHANK3. SKT-knockout (KO) mice show significant abnormalities in dendritic spine density and morphology, consistent with RhoA and Rac1 GTPase dysregulated activity. KO-derived neuronal cultures display delayed neuronal synchronization and maturation associated with glutamatergic pre- and postsynaptic impairment. Behavioral tests on KO mice reveal increased self-grooming activity and impaired motor coordination, with altered cognitive and executive functions compared to wild-type mice. Overall, SKT emerges as a key contributor to the structural and functional PSD organization, regulating synaptic function through its interactions with PSD components.
Document Type: article in journal/newspaper
File Description: ELETTRONICO
Language: English
Relation: info:eu-repo/semantics/altIdentifier/pmid/40892546; info:eu-repo/semantics/altIdentifier/wos/WOS:001566734200002; volume:44; issue:9; firstpage:1; lastpage:29; numberofpages:29; journal:CELL REPORTS; https://hdl.handle.net/10281/574543
DOI: 10.1016/j.celrep.2025.116206
Availability: https://hdl.handle.net/10281/574543; https://doi.org/10.1016/j.celrep.2025.116206
Rights: info:eu-repo/semantics/openAccess ; license:Creative Commons ; license uri:http://creativecommons.org/licenses/by/4.0/
Accession Number: edsbas.DEFB1E5E
Database: BASE