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The Jumonji-C oxygenase JMJD7 catalyzes (3S)-lysyl hydroxylation of TRAFAC GTPases

Title: The Jumonji-C oxygenase JMJD7 catalyzes (3S)-lysyl hydroxylation of TRAFAC GTPases
Authors: Markolovic, S; Zhuang, Q; Wilkins, SE; Eaton, CD; Abboud, MI; Katz, MJ; McNeil, HE; Leśniak, R; Hall, C; Struwe, WB; Konietzny, R; Davis, S; Yang, M; Ge, W; Benesch, JLP; Kessler, BM; Ratcliffe, PJ; Cockman, ME; Fischer, R; Wappner, P; Chowdhury, R; Coleman, M; Schofield, CJ
Publisher Information: Springer Nature
Publication Year: 2018
Collection: Oxford University Research Archive (ORA)
Description: Biochemical, structural, and cellular studies reveal Jumonji-C (JmjC) domain-containing 7 (JMJD7) as a 2-oxoglutarate (2OG)-dependent oxygenase catalyzing a previously unreported type of post translational modification, (3S)-lysyl hydroxylation. Crystallographic analyses reveal JMJD7 as more closely related to the JmjC hydroxylases rather than the JmjC demethylases. Biophysical and mutation studies show that JMJD7 has a unique dimerization mode, with interactions between monomers involving both N- and C-terminal regions and disulfide bond formation. A proteomic approach identifies two related members of the Translation Factor (TRAFAC) family of GTPases, Developmentally Regulated GTP Binding Proteins 1 and 2 (DRG1/2), as activity-dependent JMJD7 interactors. Mass spectrometric analyses demonstrate that JMJD7 catalyzes Fe(II)- and 2OG dependent hydroxylation of a highly-conserved lysine residue in DRG1/2; amino acid analyses reveal JMJD7 catalyzes (3S)-lysyl hydroxylation. The functional assignment of JMJD7 will enable future studies to define the role of DRG hydroxylation in cell growth and disease.
Document Type: article in journal/newspaper
Language: unknown
DOI: 10.1038/s41589-018-0071-y
Availability: https://doi.org/10.1038/s41589-018-0071-y; https://ora.ox.ac.uk/objects/uuid:62b9cef7-9260-4913-8914-346bd0d166bf
Rights: info:eu-repo/semantics/openAccess
Accession Number: edsbas.E45ADB4B
Database: BASE