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PGC-1 Coactivators Regulate MITF and the Tanning Response

Title: PGC-1 Coactivators Regulate MITF and the Tanning Response
Authors: Shoag, J; Haq, R; Zhang, M; Liu, L; Rowe, GC; Jiang, A; Koulisis, N; Farrel, C; Amos, CI; Wei, Q; Lee, JE; Zhang, J; Kupper, TS; Qureshi, AA; Cui, R; Han, J; Fisher, DE; Arany, Z
Publisher Information: //www.elsevier.com/locate/molcel; United States
Publication Year: 2013
Collection: University of Hong Kong: HKU Scholars Hub
Description: The production of pigment by melanocytes tans the skin and protects against skin cancers. UV-exposed keratinocytes secrete α-MSH, which then activates melanin formation in melanocytes by inducing the microphthalmia-associated transcription factor (MITF). We show that PPAR-γ coactivator (PGC)-1α and PGC-1β are critical components of this melanogenic system in melanocytes. α-MSH signaling strongly induces PGC-1α expression and stabilizes both PGC-1α and PGC-1β proteins. The PGC-1s in turn activate the MITF promoter, and their expression correlates strongly with that of MITF in human melanoma cell lines and biopsy specimens. Inhibition of PGC-1α and PGC-1β blocks the α-MSH-mediated induction of MITF and melanogenic genes. Conversely, overexpression of PGC-1α induces pigment formation in cell culture and transgenic animals. Finally, polymorphism studies reveal expression quantitative trait loci in the PGC-1β gene that correlate with tanning ability and protection from melanoma in humans. These data identify PGC-1 coactivators as regulators of human tanning. © 2013 Elsevier Inc. ; link_to_subscribed_fulltext
Document Type: article in journal/newspaper
Language: English
ISSN: 23201126
Relation: Molecular Cell; http://www.scopus.com/mlt/select.url?eid=2-s2.0-84872276087&selection=ref&src=s&origin=recordpage; 157; 291153; WOS:000313607300014; 145; https://hub.hku.hk/handle/10722/180756; 49
DOI: 10.1016/j.molcel.2012.10.027
Availability: https://hub.hku.hk/handle/10722/180756; https://doi.org/10.1016/j.molcel.2012.10.027
Accession Number: edsbas.E49512FA
Database: BASE