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BCL-2 family protein expression and platinum drug resistance in ovarian carcinoma.

Title: BCL-2 family protein expression and platinum drug resistance in ovarian carcinoma.
Authors: Beale, PJ; Rogers, P; Boxall, F; Sharp, SY; Kelland, LR
Source: 440 ; 436
Publisher Information: Springer Nature [academic journals on nature.com]
Publication Year: 1999
Collection: Imperial College London: Spiral
Subject Terms: Antineoplastic Agents; Apoptosis; Base Sequence; Cisplatin; DNA Damage; Drug Resistance; Neoplasm; Female; Genes; bcl-2; Humans; Molecular Sequence Data; Ovarian Neoplasms; Transplantation; Heterologous; Tumor Cells; Cultured
Subject Geographic: England
Description: The expression of the BCL-2 family proteins, BCL-2, BAX, BCL(XL) and BAK have been determined in a panel of 12 human ovarian carcinoma cell lines encompassing a wide range in sensitivity to cisplatin. Whereas BAX, BCL(XL) and BAK levels did not correlate with sensitivity, there was a statistically significant inverse correlation (r = -0.81; P = 0.002) between growth inhibition by cisplatin and BCL-2 levels. In sublines possessing acquired resistance to various platinum-based drugs or across a panel of human ovarian carcinoma xenografts, there was no consistent pattern of BCL-2 expression. Two relatively sensitive lines (A2780 and CH1) have been stably transfected with bcl-2 and bcl(XL) respectively and two relatively resistant lines (A2780cisR and SKOV-3) stably transfected with bax. Overexpression of BCL-2 in A2780 cells led to resistance to cisplatin compared to the vector control when assayed at 48 h post-drug incubation but a significant increase in sensitivity at 96 h. Relative rates of apoptosis at 48- and 96-h post-cisplatin exposure mirrored the growth inhibition. There was no significant difference in sensitivity of the pair of lines by clonogenic assay. No significant changes in chemosensitivity to a variety of DNA-damaging or tubulin-interactive agents were observed in the remaining transfected lines. Taken together, these results suggest that, in human ovarian carcinoma cells, high BCL-2 levels (either naturally occurring or through gene transfection) confers a trend towards sensitivity not resistance to platinum drugs.
Document Type: article in journal/newspaper
Language: English
Relation: British Journal of Cancer; http://hdl.handle.net/10044/1/92189
DOI: 10.1054/bjoc.1999.0939
Availability: http://hdl.handle.net/10044/1/92189; https://doi.org/10.1054/bjoc.1999.0939
Rights: © 2000 Cancer Research Campaign. This paper was modified 12 months after initial publication to switch to Creative Commons licence terms, as noted at publication References ; http://creativecommons.org/licenses/by/4.0/
Accession Number: edsbas.E4EB417
Database: BASE