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Expression of the Rap1 Guanine Nucleotide Exchange Factor, MR-GEF, Is Altered in Individuals with Bipolar Disorder

Title: Expression of the Rap1 Guanine Nucleotide Exchange Factor, MR-GEF, Is Altered in Individuals with Bipolar Disorder
Authors: Angela Bithell ¤a; Tony Hsu; Apsara K; Anearatchi ¤b; Sabine L; Ian P. Everall; Ming T; Gursharan Chana; Brenda P. Williams ¤a
Contributors: The Pennsylvania State University CiteSeerX Archives
Source: ftp://ftp.ncbi.nlm.nih.gov/pub/pmc/4b/28/PLoS_One_2010_Apr_28_5(4)_e10392.tar.gz
Publication Year: 2010
Collection: CiteSeerX
Description: In the rodent forebrain GABAergic neurons are generated from progenitor cells that express the transcription factors Dlx1 and Dlx2. The Rap-1 guanine nucleotide exchange factor, MR-GEF, is turned on by many of these developing GABAergic neurons. Expression of both Dlx1/2 and MR-GEF is retained in both adult mouse and human forebrain where, in human, decreased Dlx1 expression has been associated with psychosis. Using in situ hybridization studies we show that MR-GEF expression is significantly down-regulated in the forebrain of Dlx1/2 double mutant mice suggesting that MR-GEF and Dlx1/ 2 form part of a common signalling pathway during GABAergic neuronal development. We therefore compared MR-GEF expression by in situ hybridization in individuals with major psychiatric disorders (schizophrenia, bipolar disorder, major depression) and control individuals. We observed a significant positive correlation between layers II and IV of the dorsolateral prefrontal cortex (DLPFC) in the percentage of MR-GEF expressing neurons in individuals with bipolar disorder, but not in individuals with schizophrenia, major depressive disorder or in controls. Since MR-GEF encodes a Rap1 GEF able to
Document Type: text
File Description: application/zip
Language: English
Relation: http://citeseerx.ist.psu.edu/viewdoc/summary?doi=10.1.1.354.9607
Availability: http://citeseerx.ist.psu.edu/viewdoc/summary?doi=10.1.1.354.9607
Rights: Metadata may be used without restrictions as long as the oai identifier remains attached to it.
Accession Number: edsbas.F83242F
Database: BASE