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Genetic Diversity and Expanded Phenotypes in Dystonia: Insights From Large‐Scale Exome Sequencing

Title: Genetic Diversity and Expanded Phenotypes in Dystonia: Insights From Large‐Scale Exome Sequencing
Authors: Mirja Thomsen; Fabian Ott; Sebastian Loens; Gamze Kilic‐Berkmen; Ai Huey Tan; Shen‐Yang Lim; Ebba Lohmann; Kaja M. Schröder; Lea Ipsen; Lena A. Nothacker; Linn Welzel; Alexandra S. Rudnik; Frauke Hinrichs; Thorsten Odorfer; Kirsten E. Zeuner; Friederike Schumann; Andrea A. Kühn; Simone Zittel; Marius Moeller; Robert Pfister; Christoph Kamm; Anthony E. Lang; Yi Wen Tay; Ana Luísa deAlmeida Marcelino; Marie Vidailhet; Emmanuel Roze; Joel S. Perlmutter; Jeanne S. Feuerstein; Victor S. C. Fung; Florence Chang; Richard L. Barbano; Steven Bellows; Aparna A. Wagle Shukla; Alberto J. Espay; Mark S. LeDoux; Brian D. Berman; Stephen Reich; Andres Deik; Andre Franke; Michael Wittig; Sören Franzenburg; Jens Volkmann; Norbert Brüggemann; H. A. Jinnah; Tobias Bäumer; Christine Klein; Hauke Busch; Katja Lohmann
Source: Annals of Clinical and Translational Neurology, Vol 12, Iss 8, Pp 1648-1659 (2025)
Publisher Information: Wiley, 2025.
Publication Year: 2025
Collection: LCC:Neurosciences. Biological psychiatry. Neuropsychiatry; LCC:Neurology. Diseases of the nervous system
Subject Terms: dystonia; exome sequencing; genetic heterogeneity; pathogenic variants; Neurosciences. Biological psychiatry. Neuropsychiatry; RC321-571; Neurology. Diseases of the nervous system; RC346-429
Description: ABSTRACT Objective Dystonia is one of the most prevalent movement disorders, characterized by significant clinical and etiological heterogeneity. Despite considerable heritability (~25%), the etiology in most patients remains elusive. Moreover, understanding correlations between clinical manifestations and genetic variants has become increasingly complex. Methods Exome sequencing was conducted on 1924 genetically unsolved, mainly late‐onset isolated dystonia patients, recruited primarily from two dystonia registries (DysTract and the Dystonia Coalition). Rare variants in genes previously linked to dystonia (n = 406) were examined, confirmed via Sanger sequencing, and analyzed for segregation when possible. Results We identified 137 distinct likely pathogenic/pathogenic variants (according to ACMG criteria) across 51 genes in 163/1924 patients, including 153/1895 index patients (diagnostic yield 8.1%). The strongest predictors of a genetic diagnosis were generalized dystonia (28.6% yield) and age at onset (20.4% yield in patients with onset
Document Type: article
File Description: electronic resource
Language: English
ISSN: 2328-9503
Relation: https://doaj.org/toc/2328-9503
DOI: 10.1002/acn3.70100
Access URL: https://doaj.org/article/9ac7f4ecc7d7456ebe8b5a2a42e1860b
Accession Number: edsdoj.9ac7f4ecc7d7456ebe8b5a2a42e1860b
Database: Directory of Open Access Journals