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Memory deficits in a juvenile rat model of type 1 diabetes are due to excess 11β-HSD1 activity, which is upregulated by high glucose concentrations rather than insulin deficiency

Title: Memory deficits in a juvenile rat model of type 1 diabetes are due to excess 11β-HSD1 activity, which is upregulated by high glucose concentrations rather than insulin deficiency
Authors: Brossaud, JulieAff1, Aff2, IDs00125023059423_cor1; Bosch-Bouju, ClémentineAff1; Marissal-Arvy, NathalieAff1; Campas-Lebecque, Marie-NeigeAff1; Helbling, Jean-ChristopheAff1; Webster, Scott P.Aff3; Walker, Brian R.Aff3, Aff4; Fioramonti, XavierAff1; Ferreira, GuillaumeAff1; Barat, PascalAff1, Aff5; Corcuff, Jean-BenoîtAff1, Aff2; Moisan, Marie-PierreAff1
Source: Diabetologia: Clinical, Translational and Experimental Diabetes and Metabolism. 66(9):1735-1747
Database: Springer Nature Journals