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Reversal of proliferation deficits caused by chromosome 16p13.11 microduplication through targeting NFκB signaling: an integrated study of patient-derived neuronal precursor cells, cerebral organoids and in vivo brain imaging

Title: Reversal of proliferation deficits caused by chromosome 16p13.11 microduplication through targeting NFκB signaling: an integrated study of patient-derived neuronal precursor cells, cerebral organoids and in vivo brain imaging
Authors: Johnstone, MandyAff1, Aff2, Aff3, cor1; Vasistha, Navneet A.Aff3; Barbu, Miruna C.Aff1; Dando, OwenAff4, Aff5; Burr, KarenAff3, Aff4; Christopher, EdwardAff2; Glen, SophieAff1, Aff2; Robert, ChristelleAff6; Fetit, RanaAff3; Macleod, Kenneth G.Aff7; Livesey, Matthew R.Aff3, Aff4; Clair, David St.Aff8; Blackwood, Douglas H. R.Aff1; Millar, KirstyAff2; Carragher, Neil O.Aff7; Hardingham, Giles E.Aff4, Aff5; Wyllie, David J. A.Aff5; Johnstone, Eve C.Aff1; Whalley, Heather C.Aff1, Aff3; McIntosh, Andrew M.Aff1, Aff2, Aff3; Lawrie, Stephen M.Aff1, Aff3; Chandran, SiddharthanAff3, Aff4, Aff9, cor22
Source: Molecular Psychiatry. 24(2):294-311
Database: Springer Nature Journals